kidney damage
Introduction
Introduction Renal damage is common in liver-derived kidney damage. Hepatic-induced renal damage is also known as hepatorenal syndrome. Hepatic-induced renal damage mainly includes: 1 glomerulonephritis caused by hepatitis B virus; 2 glomerulonephritis mainly caused by IgA nephropathy caused by disorders of immunity, physiology and metabolism in liver cirrhosis; 3 severe liver In the case of insufficiency, functional acute renal failure caused by hypoperfusion of the kidney, namely hepatorenal syndrome.
Cause
Cause
Hepatic kidney damage mainly includes:
1 glomerulonephritis caused by hepatitis B virus;
2 glomerulonephritis mainly caused by IgA nephropathy caused by disorders of immunity, physiology and metabolism in liver cirrhosis;
3 severe liver dysfunction, functional acute renal failure caused by hypoperfusion of the kidney, namely liver and kidney syndrome.
Examine
an examination
Related inspection
Tumor Acoustic Contrast Urine Routine Plasma Thrombin Regulatory Protein Antigen Detection
First, medical history and symptoms
(1) Hepatitis B virus-associated nephritis is more common in children and men, patients with hepatitis B or hepatitis B virus, clinical manifestations of asymptomatic proteinuria or nephrotic syndrome, and a small number of patients may have gross hematuria.
(B) cirrhosis glomerulonephritis has a history of cirrhosis, patients can be characterized by abnormal urine test (such as microscopic hematuria), hypertension and renal insufficiency, etc., a small number of patients without clinical manifestations. Renal function deteriorates more slowly.
(3) Hepatorenal syndrome is common in the decompensation period of liver dysfunction. Most patients have certain incentives, such as excessive diuresis, massive ascites, gastrointestinal bleeding, and the use of certain nephrotoxic drugs; usually oliguria at the same time or after severe liver damage, can be days, weeks or months Progressive deterioration of renal function, often accompanied by nausea, vomiting, mental apathy and lethargy; severe cases of hepatic encephalopathy and coma. Infection, bleeding, hypotension, and liver failure are the leading causes of death.
Second, physical examination found
Hepatic disease, jaundice, spider mites, liver palm, splenomegaly, ascites, or eyelids, lower extremity edema.
Diagnosis
Differential diagnosis
Differential diagnosis of renal damage:
(1) Hepatitis B-associated nephritis needs to be differentiated from secondary glomerular diseases such as hepatitis B virus carriers and primary glomerulonephritis and lupus nephritis.
(B) renal dysfunction in the late stage of liver disease, should be differentiated from simple pre-renal azotemia, acute tubular necrosis, acute allergic interstitial nephritis.
